Experts are warning that over 100 studies have now been published that show Covid mRNA “vaccines” have triggered a deadly global “tsunami of turbo cancer.”

In 2021, mainstream science dismissed the idea that Covid “vaccines” could be linked to a surge in cancer.

But now, four years later, oncologists around the world are confronting an unsettling reality: a dramatic and unexplained rise in aggressive cancers, particularly among the young and recently vaccinated.

Professor Khayat, co-founder of France’s National Cancer Institute, once publicly rejected any link.

Today, even he admits: “We have a dramatic increase in pancreatic cancer without any idea why…

“The entire cancer research community worldwide is asking this question.”

If he is intellectually honest, he cannot rule out mass vaccination as a trigger, especially since the surge is global, sudden, and follows the unprecedented rollout of a genetic intervention never before tested on this scale.

Mathilde Debord has just published a comprehensive report in Le Point Critique, highlighting the surge in cancers among the Covid-vaccinated.

Debord’s report cites over 100 peer-reviewed studies linking mRNA injections to rapidly developing cancers.

The report outlines 17 distinct biological mechanisms by which the Covid shots initiate, accelerate, or reactivate malignant processes.

Debord warns that doctors are now “faced with a tsunami of turbo cancers, particularly among young people” who received the injections.

In May 2021, a group of French oncologists sounded the alarm, but their warnings were ignored.

Now, their grim forecasts seem prophetic.

A 2023 paper in Cureus listed 10 ways mRNA injections could promote cancer; the list has since grown to 17, backed by over 100 studies.

These aren’t fringe theories; they’re mechanisms rooted in molecular biology:

• Genomic Instability: Multiple studies suggest mRNA from the vaccine may be reverse-transcribed into human DNA, permanently altering gene regulation.
• Immune Suppression: The Spike protein interferes with p53, the “guardian of the genome,” and suppresses key immune responses that protect against cancer.
• Chronic Inflammation: Spike proteins have been found lingering in blood and organs for up to 17 months post-injection, fueling inflammation and potential tumor growth.
• Altered Microbiome: The gut microbiota—crucial for cancer resistance—is disrupted after vaccination, with a notable loss of protective bifidobacteria.
• DNA Contamination: Pfizer and Moderna shots contain unauthorized DNA fragments and SV40 sequences—oncogenic tools well-known in cancer research.

Renowned epidemiologist Nicolas Hulscher, MPH, created a breakdown of all 17 points in a report on Depord’s findings:

1. Genome Instability

mRNA may be reverse-transcribed and integrated into host DNA, triggering mutations that initiate cancer.

2. Immune Escape

The spike protein binds and inhibits tumor suppressor genes like p53 and BRCA1, shielding cancer cells from immune destruction.

3. Impaired DNA Repair Mechanism

The spike protein interferes with essential DNA repair enzymes, increasing the risk of unchecked mutations.

4. Chronic Inflammation

Lipid nanoparticles and spike protein cause long-lasting inflammation, a well-known driver of cancer.

5. Dysregulation of the Immune System

Suppression of T cells and type I interferon weakens cancer surveillance and promotes immune evasion.

6. RNA Disruption

Codon optimization disrupts microRNA networks, destabilizing cell growth regulation and apoptosis.

7. Activation of Oncogenic Pathways

The spike protein indirectly activates MAPK and PI3K/mTOR signaling, fueling tumor growth and metastasis.

8. Tumor Microenvironment Alteration

Lipid nanoparticles accumulate in tumors, enhancing permeability and potentially accelerating cancer spread.

9. Awakening Dormant Cancers

Post-vaccination inflammation and immune disruption may trigger recurrence in patients previously in remission.

10. Alteration of Immune Surveillance

Modified mRNA blocks toll-like receptors, making tumor cells “invisible” to the immune system.

11. Frameshift Errors

The synthetic mRNA sometimes produces unintended, aberrant proteins, contributing to oncogenic risk.

12. Multiple Injections

Repeated doses exhaust the immune system and drive class switching to IgG4, promoting tolerance to tumors.

13. DNA Contamination

Residual plasmid DNA found in vaccine vials is replication-competent and could integrate into host genomes.

14. Oncogenic SV40 DNA Sequences

SV40 promoter sequences in Pfizer vials may facilitate genome insertion—this same element is used to induce tumors in lab animals.

15. Deregulation of the Renin-Angiotensin System (RAS)

Spike-induced AT1R activation fosters oxidative stress and uncontrolled cell proliferation.

16. Destruction of the Microbiota

17. Increased Resistance to Treatments

Spike exposure prolongs cancer cell survival during chemotherapy, possibly driving treatment resistance.

Each point alone would warrant serious concern.

Together, they paint a devastating picture.

Pathologists are now reporting that cases of “turbo cancers” are skyrocketing.

These cancers are aggressive tumors growing at an unprecedented speed.

Patients in remission are relapsing weeks after booster shots.

In Australia, a major clinical trial investigating the link between mRNA “vaccines” and cancer was abruptly halted, as Slay News reported.

Alarmingly, officials ordered all biological samples from the trials to be destroyed.

It comes after ivermectin was maligned and blocked from being used as a treatment for Covid.

Ivermectin is safe, Nobel Prize–winning, and proven in anti-cancer research.

Meanwhile, experimental injections with no long-term safety data were forced upon entire populations.

The cancer explosion is real, and governments around the world know it.

A growing number of researchers are asking questions.

But instead of investigating, authorities are deflecting.

For the millions suffering, and for those who complied under coercion, these questions are not theoretical.

They are urgent, deeply personal, and demand answers.

Source:  https://slaynews.com/news/over-100-studies-prove-covid-vaccines-caused-tsunami-turbo-cancer/

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A world-renowned immunologist is sounding the alarm after his peer-reviewed research uncovered a devastating surge in a deadly heart condition among people who received Covid mRNA “vaccines.”

Dr. János Szebeni, an internationally recognized European immunologist, issued a stomach-churning warning to the world after confirming that the injections have caused a staggering 115,100% surge in myocarditis.

Myocarditis is a “silent killer” at the center of the sudden deaths crisis that emerged after the Covid injections were rolled out for public use in early 2021.

The mind-blowing discovery was revealed in a sweeping peer-reviewed study by Dr. Szebeni, a professor at Hungary’s Semmelweis University and Sungkyunkwan University and a leading immunotoxicology researcher at SeroScience.

Dr. Szebeni is calling for a critical reevaluation of mRNA injections’ classification as “vaccines.”

Source:  https://slaynews.com/news/deadly-heart-condition-surges-115100-covid-mrna-vaccinated/

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My latest show with Hope and Tivon shares the incredible science of transforming a natural human being into a 100% synthetic one; down to each individual cell. It seems like science fiction until you study the white papers and understand the research. Its harder to believe that anyone would actually want to work on this type of research.

The show is really long, so it was split into 2 parts. Part one you can see with the link below (on many different platforms) and part 2 is uploaded here for all Substack subscribers.

Description: Hope and Tivon rejoin the program to discuss the new research and direction they are seeing in the synthetic biology and IEEE world. They have been reading and analyzing books and white papers to track where the researchers and projects are heading. They also share hopeful news on how to break down unwanted synthetic materials in your body. You can purchase the EMF kit discussed during the program at https://www.ftwproject.com/product/brighteon-university-emf-protection-sample-kit/

Link to Part 1: Future Humans 100% Synthetic down to the individual Cells w/ Hope & Tivon – Part 1

Part 2 is uploaded here to Substack.

Source:  https://sarahwestall.substack.com/p/future-humans-100-synthetic-down?utm_source=podcast-email&publication_id=1456754&post_id=150332058&utm_campaign=email-play-on-substack&utm_content=watch_now_button&r=1h19iv&triedRedirect=true&utm_medium=email

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My latest show with Hope and Tivon shares the incredible science of transforming a natural human being into a 100% synthetic one; down to each individual cell. It seems like science fiction until you study the white papers and understand the research. Its harder to believe that anyone would actually want to work on this type of research.

The show is really long, so it was split into 2 parts. Part one you can see with the link below (on many different platforms) and part 2 is uploaded here for all Substack subscribers.

Description: Hope and Tivon rejoin the program to discuss the new research and direction they are seeing in the synthetic biology and IEEE world. They have been reading and analyzing books and white papers to track where the researchers and projects are heading. They also share hopeful news on how to break down unwanted synthetic materials in your body. You can purchase the EMF kit discussed during the program at https://www.ftwproject.com/product/brighteon-university-emf-protection-sample-kit/

Link to Part 1: Future Humans 100% Synthetic down to the individual Cells w/ Hope & Tivon – Part 1

Part 2 is uploaded here to Substack.

Source:  https://sarahwestall.substack.com/p/future-humans-100-synthetic-down?utm_source=podcast-email&publication_id=1456754&post_id=150332058&utm_campaign=email-play-on-substack&utm_content=watch_now_button&r=1h19iv&triedRedirect=true&utm_medium=email

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Forbidden Tech Book: www.forbiddentech.website

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Research led by Oregon State University demonstrates that a single individual can effectively oversee a “swarm” of more than 100 autonomous ground and aerial robots. This achievement, funded by the Defense Advanced Research Project Agency’s OFFSET program, has vast implications across various sectors, including firefighting, disaster response, and package delivery services. Julie A. Adams from OSU’s College of Engineering emphasizes the potential for scaled deployment of delivery drones, advocating for a centralized supervisory approach.

The study, published in Field Robotics, highlights the initial steps toward understanding the feasibility of such systems, offering promising insights into future applications. The four-year project involved deploying swarms of up to 250 autonomous vehicles in urban environments, integrating various swarm tactics and autonomy technologies. Adams, as co-principal investigator, led the development of system infrastructure and a virtual reality interface named I3.

This interface enables commanders to control the swarm effectively, akin to a quarterback in American football. Physiological sensors monitored commanders’ workload levels during multiday field exercises, demonstrating the system’s reliability and adaptability under challenging conditions. Despite occasional workload thresholds, commanders successfully completed missions, showcasing the system’s effectiveness in real-world scenarios.

Source:  https://www.youtube.com/watch?v=YJFLRNU1vVI

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The post HOW ONE PERSON CAN SUPERVISE 100 UNMANNED AUTONOMOUS VEHICLES appeared first on HopeGirl Blog.

The first time TruNews visited Barcelona in 2017, we heard about the Global Brain. The target date to complete the global brain was 2030. The last time we visited Barcelona was in 2019. We witnessed in real time the microchipping of a human being, and the instant transfer of money from one bank to another by scanning a microchipped man’s arm with a phone. What is the event? It is Mobile World Congress Barcelona 2024. Over 100,000 communication and technology professionals will attend from many nations.

It is the official global conference of the GMSA, the mobile phone industry’s trade association for 750 mobile phone operators. We have not attended a Mobile World Congress for five years. Why are we returning in 2024? If you want to know where technology is going, this is one of the best conferences in the world to attend. Today, we will look back to that first Mobile World Congress that TruNews attended in 2017 where we heard about the coming Global Brain.

Source:  https://www.brighteon.com/567b2ae2-6dc0-4e4c-b396-ab4c50705d98

Bitchute: https://www.bitchute.com/channel/YBM3rvf5ydDM/

Gab: https://gab.com/hopegirl

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EMF Protection Products: www.ftwproject.com

QEG Clean Energy Academy: www.cleanenergyacademy.com

Forbidden Tech Book: www.forbiddentech.website

The post Global Brain & 6G – TruNews Headed to Barcelona Over 100,000 communication and technology professionals will attend from many nations appeared first on HopeGirl Blog.

Lethal Infection of Human ACE2-Transgenic Mice Caused by SARS-CoV-2-related Pangolin Coronavirus GX_P2V(short_3UTR)

Dear Editor

Two SARS-CoV-2-related pangolin coronaviruses, GD/2019 and GX/2017, were identified prior to the COVID-19 outbreak (1, 2). The respective isolates, termed pCoV-GD01 and GX_P2V, were cultured in 2020 and 2017, respectively (2, 3). The infectivity and pathogenicity of these isolates have been studied (4–6). The pCoV-GD01 isolate, which has higher homology with SARS-CoV-2, can infect and cause disease in both golden hamsters and hACE2 mice (4). In contrast, while GX_P2V can also infect both species, it does not appear to cause obvious disease in these animals (5, 6). We previously reported that the early passaged GX_P2V isolate was actually a cell culture-adapted mutant, named GX_P2V(short_3UTR), which possesses a 104-nucleotide deletion at the 3’-UTR (6). In this study, we cloned this mutant, considering the propensity of coronaviruses to undergo rapid adaptive mutation in cell culture, and assessed its pathogenicity in hACE2 mice. We found that the GX_P2V(short_3UTR) clone can infect hACE2 mice, with high viral loads detected in both lung and brain tissues. This infection resulted in 100% mortality in the hACE2 mice. We surmise that the cause of death may be linked to the occurrence of late brain infection.

The GX_P2V(short_3UTR) mutant, initially isolated from the early passages of the GX_P2V sample (6), and the GX_P2V virus itself, have not been studied in terms of their adaptive mutations in cell cultures. To obtain a genetically homogenous clone for animal experiments, we cloned the passaged mutant through two successive plaque assays. Eight viral clones were chosen for next-generation sequencing (National Genomics Data Center of China, GSA: CRA014225). These clones, when compared with the genome of the original mutant (6), all shared four identical mutations: ORF1ab_D6889G, S_T730I, S_K807N, and E_A22D (Supporting Information, Table S1). Clone 7, named as GX_P2V C7, was randomly selected for the evaluation of viral pathogenicity in hACE2 mice (Figure 1A). The hACE2 mouse model expressing human ACE2 under control of the CAG promoter was developed using random integration technology by Beijing SpePharm Biotechnology Company.

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Figure 1:Characterization of a lethal infection model in human ACE2-transgenic mice caused by the attenuated SARS-CoV-2-related pangolin coronavirus GX_P2V C7.

A Mutations in GX_P2V C7 compared to the GX_P2V(short_3UTR) isolate (NCBI accession number: MW532698). The four identical mutations are shown in bold. B Survival of hACE2 transgenic mice following intranasal infection with GX_P2V C7 (n = 4), inactivated GX_P2V C7 (i-C7, n = 4), and mock infection (n = 4). The number of deceased mice on each specific day is annotated on the left of the survival curve. C Percentage of initial weight of hACE2 transgenic mice after intranasal infection with GX_P2V C7 (n = 4), i-C7 (n = 4), and mock infection (n = 4). The statistical significance of the differences between mock-infected (n = 4, blue dots) and GX_P2V C7-infected (n = 4, red dots) or i-C7-infected mice (n = 4, orange dots) at 6 or 7 dpi are shown. The error bars represent the means ± SDs. D Quantification of GX_P2V N gene copies in heart, liver, spleen, lung, kidney, tongue, intestine, stomach, trachea, brain, eye, and turbinate homogenates at 3- and 6-day post-infection (dpi) (n = 4 per group). The limit of detection (LOD) for viral RNA loads in the original samples was Log₁₀[10² copies/mg]. The error bars represent the means of Log₁₀[copies/mg] ± SDs. The significances of the comparisons in the lung, brain, and turbinate are shown. E Infectious viral titers in lung, brain, eye, and turbinate homogenates were measured by plaque forming assay at 3 and 6 dpi (n = 4 per group). The statistical significance of the differences in the lung, brain, and turbinate are shown. The error bars represent means of Log₁₀[pfu/mL] ± SDs. F, G Hematoxylin and eosin (H&E) staining and immunohistochemical (IHC) staining with an anti–SARS-CoV-2 N-specific antibody (SARS-CoV-2) revealed viral antigen–positive cells (brown) in the lung (F) and brain (G), as shown at high magnification in the inset. Scale bars, 500 μm (F) and 1 mm (G), respectively. *P < 0.05, **P < 0.01, ***P < 0.001, ****P < 0.0001, P > 0.05, not significant (ns); two-way ANOVA followed by Sidak’s multiple comparison test.

We initially assessed whether GX_P2V C7 could cause disease in hACE2 mice by monitoring daily weight and clinical symptoms. A total of four 6 to 8-week-old hACE2 mice were intranasally infected with a dosage of 5×10⁵ plaque-forming units (pfu) of the virus. Four mice inoculated with inactivated virus and four mock-infected mice were used as controls. Surprisingly, all the mice that were infected with the live virus succumbed to the infection within 7-8 days post-inoculation, rendering a mortality rate of 100% (Figure 1B). The mice began to exhibit a decrease in body weight starting from day 5 post-infection, reaching a 10% decrease from the initial weight by day 6 (Figure 1C). By the seventh day following infection, the mice displayed symptoms such as piloerection, hunched posture, and sluggish movements, and their eyes turned white. The criteria for clinical scoring of the mice and the daily clinical scores post-infection with GX_P2V C7 are provided in the Supporting Information, Figure S1.

We then evaluated the tissue tropism of GX_P2V C7 in hACE2 mice. Using the infection method described above, eight hACE2 mice were infected, eight mice were inoculated with inactivated virus, and another eight mock-infected mice were used as controls. The organs of four randomly selected mice in each group were dissected on days 3 and 6 post-infection for quantitative analysis of viral RNA and titer. We detected significant amounts of viral RNA in the brain, lung, turbinate, eye, and trachea of the GX_P2V C7 infected mice (Figure 1D), whereas no or a low amount of viral RNA was detected in other organs such as the heart, liver, spleen, kidneys, tongue, stomach, and intestines. Specifically, in lung samples, we detected high viral RNA loads on days 3 and 6 post-infection, with no significant difference between these two time points (∼ 6.3 versus ∼ 5.8 Log₁₀[copies/mg]). In brain samples, on day 3 post-infection, viral RNA was detected in all four infected mice, with an average value of 5.4 Log₁₀[copies/mg]. Notably, by day 6 post-infection, we detected exceptionally high viral RNA loads (∼ 8.5 Log₁₀[copies/mg]) in the brain samples from all four infected mice (Figure 1D). On days 3 and 6 post-infection, the viral RNA loads in the turbinate were similar, approximately 4.1 and 3.9 Log₁₀[copies/mg], respectively. The viral RNA loads in the trachea and eyes of the mice surpassed the limit of detection only on day 6 post-infection, with values of 2.6 and 3.8 Log₁₀[copies/mg], respectively. Regarding the infectious viral titers, lung tissues at day 3 post-infection had a value of ∼ 1.8 Log₁₀[pfu/mg], which decreased to ∼ 0.5 Log₁₀[pfu/mg] by day 6. Importantly, the highest infectious titers were detected in the brain on day 6, which was significantly greater than that on day 3 (∼ 0.9 vs ∼ 4.8 Log₁₀[pfu/mg]) (Figure 1E). Additionally, there were no significant differences in the infectious titers in the turbinate between day 3 (∼ 0.9 Log₁₀[pfu/mg]) and day 6 (∼ 1.2 Log₁₀[pfu/mg]) (Figure 1E). By day 6, approximately 2.0 Log₁₀[pfu/mg] was detected in the eyes of two mice. Neither inactivated GX_P2V C7 nor mock infection caused death or any clinical symptoms in the mice (Figure 1B-C and Supporting Information, Figure S2). In summary, in the mice infected with live virus, the viral load in the lungs significantly decreased by day 6; both the viral RNA loads and viral titers in the brain samples were relatively low on day 3, but substantially increased by day 6. This finding suggested that severe brain infection during the later stages of infection may be the key cause of death in these mice.

To determine the mechanisms underlying GX_P2V C7-induced death in hACE2 mice, we examined the pathological changes, presence of viral antigens, and cytokine profiles in the lung and brain tissues of the mice on days 3 and 6 post-infection(Figure 1F-G, and Supporting Information, Figure S3 and S4). On both days, compared to those of control mice, the lungs of infected mice showed no significant pathological alterations, with only minor inflammatory responses due to slight granulocyte infiltration (Figure 1F). On day 3 post-infection, shrunken neurons were visible in the cerebral cortex of the mice. By day 6, in addition to the shrunken neurons, there was focal lymphocytic infiltration around the blood vessels, although no conspicuous inflammatory reaction was observed (Figure 1G). Upon staining for viral nucleocapsid protein via immunohistochemistry, viral antigens were detected in both the lungs and brains on days 3 and 6 post-infection, with extensive viral antigens notably present in the brain on day 6 (Figure 1F-G). These findings align with the viral RNA load assessments in the lung and brain tissues (Figure 1D). We also performed a Luminex cytokine assay to detect 31 cytokines/chemokines in the lung and brain tissues of the mice (Supporting Information, Figure S3 and S4). Consistent with the pathological findings, there were slight increases or decreases in the levels of many cytokines/chemokines in lung and brain tissues compared to those in control tissues, but the levels of key inflammatory factors, such as IFN-γ, IL-6, IL-1β, and TNF-α, did not significantly change. In brief, these analyses revealed that GX_P2V C7 infection in hACE2 mice did not lead to severe inflammatory reactions, a finding that aligns with previous reports by Zhengli Shi’s group using GX_P2V infection in two different hACE2 mouse models (5), as well as our own findings in the golden hamster model (6).

To the best of our knowledge, this is the first report showing that a SARS-CoV-2-related pangolin coronavirus can cause 100% mortality in hACE2 mice, suggesting a risk for GX_P2V to spill over into humans. Our findings are evidently inconsistent with those of Zhengli Shi et al. (5), who tested the virulence of GX_P2V in two different hACE2 mouse models. It is important to note that we did not isolate the wild-type GX_P2V strain. The study by Zhengli Shi et al tested the GX_P2V(short_3UTR) variant that we reported. However, the adaptative evolutionary changes of this variant during their laboratory culture remain understudied. In fact, according to additional infection experiments, the uncloned GX_P2V(short_3UTR) also resulted in 100% mortality in hACE2 mice. Due to the propensity of coronaviruses to undergo adaptive mutation during passage culture, we cloned and analyzed mutations in GX_P2V(short_3UTR), focusing specifically on the pathogenicity of the cloned strains. The high pathogenicity mechanism of GX_P2V C7 in hACE2 mice, in the absence of the wild-type GX_P2V control, requires further investigation. Compared to the original sequence of GX_P2V(short_3UTR), GX_P2V C7 has two amino acid mutations in the spike protein. Given the close relationship between coronavirus virulence and spike protein mutations (7), it is possible that GX_P2V C7 has undergone a virulence-enhancing mutation. However, it is important to note that our hACE2 mouse model may be relatively unique. The company has not yet published a paper on this hACE2 mouse model, but our results suggest that hACE2 may be highly expressed in the mouse brain. Additionally, according to the data provided by the company, these hACE2 mice have abnormal physiology, as indicated by relatively reduced serum triglyceride, cholesterol, and lipase levels, compared to those of wild-type C57BL/6J mice. In summary, our study provides a unique perspective on the pathogenicity of GX_P2V and offers a distinct alternative model for understanding the pathogenic mechanisms of SARS-CoV-2-related coronaviruses.

Source:  https://www.biorxiv.org/content/10.1101/2024.01.03.574008v1.full

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100+ makers, designers, engineers, scientists, and geeks gathered at POC21 and spent five weeks developing 12 sustainable lifestyle technologies.

When a group of talented and committed individuals puts their minds together to overcome our current “destructive consumer culture” by designing and building open-source solutions for a more sustainable future, great things can happen, as illustrated by the results of the participants of last year’s POC21 (Proof of Concept) camp.

In order to make progress toward a more livable future, it’s important to have a multi-pronged approach, with viable and affordable solutions developed for such basic needs as energy, food, transportation, and not just on a large scale, but also on a personal scale. Sure, it’s great that we’re seeing higher rates of solar adoption, and more innovations in low-carbon transport methods, but if all of these solutions are top-down (or capital-intensive, as a home solar array or a new EV is), then they will continue to be limited in their scope and their effects, due to the financial restraints that many of us come up against.

The folks behind the POC21 project are taking a stab at building alternative solutions that could have a significant impact on people’s lives, and are focusing their efforts on open source sustainable products that will lend themselves to “distributed fabrication” and collaborative production.

“POC21 is an international innovation community that started as an innovation camp. The camp brought together 100+ makers, designers, engineers, scientists and geeks. Late summer 2015, we have joined forces in a stunning French castle to prototype the fossil free, zero waste society. Our ultimate goal was to overcome the destructive consumer culture and make open-source, sustainable products the new normal. Over the course of 5 weeks we developed 12 sustainable lifestyle technologies and built an international community of innovators and supporters, that continues to grow.”

From a group of about 200 applicants, POC21 selected “the 12 most-promising open source products” in the fields of food, energy, housing, mobility, and communications, and the five-week innovation camp that was held from August 15th to September 20th brought together some 100 participants “create a proof of concept for a sustainable tomorrow.”

The final 12 projects/products can be seen on the POC21 website, some of which we’ve covered before, such as the $30 DIY wind turbine and the Aker urban ag kits, but there are also compelling projects for DIY solar (both PV and solar thermal), cargo bikes, 3D-printed water filters, home food production, and a pedal-powered tractor. Most of these projects also have tutorials posted online, so it’s a great jumping-off point for the doers and makers who want to get hands-on with sustainable projects.

By Derek Markham (@derekmarkham)

Derek is a digital dad with an analog streak, as comfortable with a smartphone as he is with pencil and paper. He’s got a soft spot for babies, bicycles and bouldering, and is the Head Shoveler at his mini-farm startup in New Mexico. You can also find him letting it all hang out on Twitter, Facebook, and NaturalPapa.

(Source:  treehugger.com; January 20, 2016; http://tinyurl.com/honkvcm)

The post 100+ makers eco-hack the future with open-source prototypes appeared first on HopeGirl Blog.

Free Energy can be a dirty business, and it’s time to clean out the closet! In this show we cover cold fusion, and inventors Pons and Fleishman, Bruce De Palma, and Stanley Meyer.  Inventions covered are the N Machine and the E-Cat.

We’ll also discuss the free energy mafia and archetypes, JTRIG Internet Trolling, Top Free Energy Sites, Eugene Mallove, Steven E. Jones,  Bearden, Edgar Mitchell, Stephen Greer, Sterling Allan, Mark Dansie and free energy related deaths.

To be covered in our “Dirty Games” Episode of the Peoples Free Energy Show. Airs Live at 9PM GMT /

View Archive Here:

 Link to Power Point Presentation for the show:
https://www.hopegirlblog.com/wp-content/uploads/2016/02/Peoples-Free-Energy-Show-Slide-Episode-2-Dirty-Games.pptx 

List of Content Included in Slide: (In order)
Free Energy Mafia
Vocabulary: Cold Fusion
Pons & Fleishmann and Cold Fusion
Steve Jones & Cold Fusion
Cold Fusion Summary
Bruce De Palma’s N- Machine
Death of Bruce De Palma
Dr. Eugene Mallove
Stan Meyers Cel
Andrea Rossi E-Cat
E-Cat Cold Fusion & LENR
Deaths Related to Free Energy
Stan Meyers Death
How do we get Access to Free Energy Devices
Dr.Steven Greer and the $100K  STAR Challenge
JTRIG
Internet Trolling
Confessions of a Paid Shill
Free Energy Archetypes

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